On his blog, Mercola.com, Dr. Joseph Mercola examines the idea that the COVID-19 vaccination could be harming immune systems, and allowing infections like shingles to occur more often than normal in those receiving them. Mercola bases his analysis on a recently published case study in the Journal of Cutaneous Immunology and Allergy. He writes:
According to a recent case study1 published in the Journal of Cutaneous Immunology and Allergy, persistent post-jab shingles, aka herpes zoster, an infection caused by the varicella zoster virus, was associated with the presence of COVID jab spike protein in the affected skin. As explained by the authors:2
“Since the campaign of vaccination against COVID-19 was started, a wide variety of cutaneous adverse effects after vaccination has been documented worldwide. Varicella zoster virus (VZV) reactivation was reportedly the most frequent cutaneous reaction in men after administration of mRNA COVID-19 vaccines, especially BNT162b2.
A patient, who had persistent skin lesions after BNT162b2 vaccination for … over 3 months, was investigated for VZV virus and any involvement of vaccine-derived spike protein … Strikingly, the vaccine-encoded spike protein of the COVID-19 virus was expressed in the vesicular keratinocytes and endothelial cells in the dermis.”
COVID Jab Impairs Your Immune Function
The researchers speculate that the COVID jab may induce persistent shingles reactivation by “perturbing the immune system.” How your immune system is perturbed by the COVID shots is the topic of MIT researcher Stephanie Seneff’s paper3 “Innate Immune Suppression by SARS-CoV-2 mRNA Vaccinations: The Role of G-quadruplexes, Exosomes and MicroRNAs,” co-written with Drs. Peter McCullough, Greg Nigh and Anthony Kyriakopoulos.
In it, they describe how the COVID shots suppress your innate immune system by inhibiting the type-1 interferon pathway, which is the first-stage response to all viral infections.
When a cell is invaded by a virus, it releases type-1 interferon alpha and beta. Both of these molecules act as signaling molecules that tell the cell it’s been infected. That, in turn, launches the immune response and gets it going early in the viral infection.
Type-1 interferon also keeps latent viruses in check, so if your interferon pathway is suppressed, latent viruses can start to emerge. The U.S. Vaccine Adverse Event Reporting System (VAERS) database reveals many who have been jabbed report these kinds of infections. Regulators in the European Union are also warning that repeat COVID shots can weaken overall immunity.4
How is type-1 interferon suppressed by the jab? It’s suppressed because type-1 interferon responds to viral RNA, and viral RNA is not present in the COVID shot. The RNA is modified to look like human RNA, so the interferon pathway is not triggered. Worse, the interferon pathway is actively suppressed by the microRNA in the shot, which limits your ability to fight off all viruses. It also opens the door for latent viruses to reactivate.
In the case study above, the patient had received one dose of Pfizer’s mRNA shot 13 days before the shingles outbreak and a second dose eight days after, causing the researchers to suspect there was an association between the outbreak and the shots.
COVID Jab and Shingles in the Immunocompromised
Similarly, a study5,6 published in April 2021 detailed the cases of six patients with autoimmune inflammatory rheumatic diseases who developed shingles shortly after their Pfizer jabs.
“The safety profile of mRNA-based vaccines in patients with autoimmune inflammatory rheumatic diseases (AIIRD) is unknown,” the authors noted.7 “The objective of this report is to raise awareness of reactivation of herpes zoster (HZ) following the BNT16b2 mRNA vaccination in patients with AIIRD.”
The study found herpes infection occurred in 1.2% of AIIRD patients who got the shot (six out of 491), compared to none among controls. Five of them developed shingles for the first time in their life a short time after their first dose of Pfizer. One got it after her second dose.
As noted by the authors, immunosuppressed patients, including patients with AIIRD, have been prioritized for the COVID jab, even though immunosuppressed patients were excluded from the clinical trials. The fact that 1.2% of immunocompromised patients developed shingles, whereas none of the healthy controls suffered this fate, shows you just how important it is not to confine clinical trials to the healthiest among us.
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